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We report a case of an acute ST-segment elevation myocardial infarction in a patient with conventional cardiac risk factors using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person also had polycythemia, likely secondary to high-dose testosterone. The affected person underwent profitable percutaneous intervention of the best coronary artery. Physicians and patients should pay consideration to the scientific consequences of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996). Moreover, a part of endothelial dysfunction has been proposed, which may contribute to abnormal vessel reactivity.

Moreover, testosterone has been used in the treatment of reduced muscle mass and bone density in addition to anemia. The position testosterone might play in the improvement of cardiovascular disease has increasingly turn out to be the topic of current curiosity . Anabolic androgenic steroid use has increased over the previous decade, regardless of its potential for opposed effects. They continue to be used by Olympic level athletes, and have even unfold to high school level athletes, noncompetitive bodybuilders and recreational athletes (Sullivan et al 1998; Dhar et al 2007).

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• The left anterior artery demonstrates a 95% stenosis after the primary septal branch.
• Anabolic androgenic steroid use has increased over the previous decade, despite its potential for adverse effects.
• Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).

However, testosterone in physiologic doses could even be helpful in sufferers with angina , have greater ischemic thresholds and improved quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and increases coronary blood circulate in men with established coronary artery disease . Therefore, an interaction of endogenous elements of the patient’s risk profile in addition to the testosterone dosing may be issues of whether or not opposed events occur. Erythrocytosis has been reported as a potential effect of testosterone, which can be beneficial in patients with anemia. The greatest risk of erythrocytosis occurs with injectable types of testosterone compared with transdermal preparations, especially in supraphysiologic doses. An enhance of as much as 44% of the baseline hematocrit has been reported .

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Unfortunately, the info on the use of excessive dose anabolic steroids in humans are primarily offered as case stories or small research that lack adequate control groups. In addition, the information out there within the literature doesn’t account for the steroid type or dose, as neither could also be known. Patients also may be taking concomitant stimulants, similar to ephedra, which may confound knowledge and the potential for adverse occasions. It is likely that the frequency of cardiac events is underreported in the medical literature. Supraphysiologic doses of anabolic-androgenic steroids , which include greater than 30 natural and synthetic derivatives of testosterone, could cause a wide spectrum of clinical sequalae.

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